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In the section Metabolism 101, we learned that if you lose weight from lean tissue, this slows
your metabolism, making weight loss and weight control more difficult.

In the section Metabolism 201, you learned that if you try and lose weight too fast, your
body thinks you are starving and consumes lean tissues and fat tissues. This slows our
metabolism, making weight loss and weight control more difficult.

In this section, we shall learn how the food industry engineered foods to make you hungry
by clogging insulin receptors. This helps them sell more food. Here is how it works.

When we eat food, the digestive system converts the energy in the food into blood sugar.
This process of the body turning food into blood sugar normally happens very slowly and
gradually. When the pancreas senses a rise in blood sugar, and appropriate amount insulin
is secreted by the pancreas. The insulin takes the blood sugar to our cells, connects to an
insulin receptor site on the cell membrane, and opens a door that allows the blood sugar to
enter the cell. This is how our cells get the energy they need to live.

The cell use fats as building blocks for the cell membranes. Trans fats make terrible
building blocks. Simply put, a diet high in trans fats creates insulin receptors that
malfunction on lean tissues. Unfortunately, insulin receptors on fat tissues take longer to
malfunction from using trans fats as building blocks, so this kind of food source starves lean
tissues and nourishes fat tissues. Omega 3 fats like flax and fish oil make great cell
membranes. For this reason, we recommend you eliminate trans fats and supplement
Omega 3 fats. We will provide instruction on how to supplement Omega 3 fats in the section
on Supplements.

The other way to clog insulin receptors is to engineer food to spike blood sugar. If you eat
complex carbohydrates like fruits, vegetables, and whole grains, your blood sugar rises very
slightly and very gradually. Just a bit of insulin is secreted by the pancreas. If you eat
refined sugars, flours, and starches, your blood sugar spikes very rapidly. The pancreas
was never designed to cope with these engineered foods, so the organ overreacts and
secretes too much insulin. This over production of insulin causes your blood sugar to crash.
You then crave the kind of foods that get blood sugar up fast, so you go for another helping
of these refined sugars, flours, and starches. Ever wonder why you can eat cookie after
cookie after cookie?

Eventually, the cell membranes develop a resistance to this constant overproduction of
insulin. The insulin receptor sites start to malfunction. The cells do not recognize the
malfunction is in the cell membrane. The cells think there is not enough blood sugar, so it
signals the brain to secrete chemicals to stimulate appetite. The pancreas senses the ever
rising levels of blood sugar spikes and makes more and more insulin. Insulin levels keep
rising, causing insulin receptor sites to become more resistant, causing insulin levels to rise
even higher, causing the body to store the blood sugar as fat tissue.

The body keeps blood sugar levels normal for a while because the overproduction of insulin
forces the blood sugar into fat tissue, but eventually, even the fat tissue becomes resistant
to insulin and blood sugar levels begin to rise to abnormal levels. Your doctor calls this
pre-Diabetes. Eventually the pancreas starts to burn out and blood sugar levels rise even
higher. Your doctor calls this Diabetes Type II. When your pancreas gives out altogether,
your doctor prescribes insulin in the form of a drug to keep your blood sugar normal. Your
doctor calls this Diabetes Type I. The kind of insulin that you use as a drug makes weight
control almost impossible. Ask anyone you know who is taking insulin prescribed by their
doctor. They will very likely tell you that they are gaining weight. Eventually though, even
using the insulin prescribed by your doctor begins to fail, and your body slowly starves to
death.

In the next section Metabolism 401, you will learn how to repair metabolism.